Search results for " Nicotinic"

showing 10 items of 105 documents

Intra-neuronal Competition for Synaptic Partners Conserves the Amount of Dendritic Building Material

2017

Brain development requires correct targeting of multiple thousand synaptic terminals onto staggeringly complex dendritic arbors. The mechanisms by which input synapse numbers are matched to dendrite size, and by which synaptic inputs from different transmitter systems are correctly partitioned onto a postsynaptic arbor, are incompletely understood. By combining quantitative neuroanatomy with targeted genetic manipulation of synaptic input to an identified Drosophila neuron, we show that synaptic inputs of two different transmitter classes locally direct dendrite growth in a competitive manner. During development, the relative amounts of GABAergic and cholinergic synaptic drive shift dendrit…

0301 basic medicineDendritic spinePresynaptic TerminalsBiologyReceptors NicotinicArticleSynapse03 medical and health sciencesDendrite (crystal)Calcium Channels T-Type0302 clinical medicinePostsynaptic potentialSynaptic augmentationmedicineAnimalsDrosophila ProteinsCalcium Signalinggamma-Aminobutyric AcidNeuronsNeuronal PlasticityGeneral NeuroscienceDendritesReceptors GABA-AAcetylcholine030104 developmental biologySynaptic fatiguemedicine.anatomical_structurenervous systemSynaptic plasticitySynapsesDrosophilaNeuronNeuroscience030217 neurology & neurosurgery
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Closed-Locked and Apo-Resting State Structures of the Human α7 Nicotinic Receptor: A Computational Study

2018

International audience; Nicotinic acetylcholine receptors, belonging to the Cys-loop super-family of ligand-gated ion channels (LGICs), are membrane proteins present in neurons and at neuromuscular junctions. They are responsible for signal transmission, and their function is regulated by neurotransmitters, agonists and antagonists drugs. A detailed knowledge of their conformational transition in response to ligand binding is critical to understand the basis of ligand-receptor interaction, in view of new pharmacological approaches to control receptor activity. However, the scarcity of experimentally derived structures of human channels makes this perspective extremely challenging. To contri…

0301 basic medicinealpha7 Nicotinic Acetylcholine ReceptorProtein ConformationGeneral Chemical EngineeringMolecular Dynamics SimulationLibrary and Information Sciences03 medical and health sciencesMolecular dynamics0302 clinical medicineHumansHomology modelingReceptorIon channelAcetylcholine receptor[SDV.BBM.BS]Life Sciences [q-bio]/Biochemistry Molecular Biology/Structural Biology [q-bio.BM]Protein StabilityChemistryWaterHydrogen BondingGeneral ChemistryLigand (biochemistry)molecular dynamicsComputer Science Applications[SDV.BBM.BP]Life Sciences [q-bio]/Biochemistry Molecular Biology/BiophysicsTransmembrane domain030104 developmental biologyNicotinic agonistBiophysics[INFO.INFO-BI]Computer Science [cs]/Bioinformatics [q-bio.QM]Conotoxinsligand gated ion channel030217 neurology & neurosurgery
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A possible desensitized state conformation of the human α7 nicotinic receptor: A molecular dynamics study

2017

International audience; The determination of the conformational states corresponding to diverse functional roles of ligand gated ion channels is subject of intense investigation with various techniques, from X-rays structure determination to electrophysiology and computational modeling. Even with a certain number of structures becoming recently available, only few major structural features distinguishing conductive open channel from the non conductive resting protein have been highlighted, while high-resolution details are still missing. The characterization of the desensitized conformation(s) is even more complex, and only few specific characteristics have been identified. Furthermore, exp…

0301 basic medicinealpha7 Nicotinic Acetylcholine ReceptorStereochemistryPyridinesBiophysicsMolecular Dynamics SimulationBiochemistry03 medical and health sciencesMolecular dynamicsmedicineHumansHomology modelingnicotinic receptor epibatidine molecular dynamics inactive stateIon channel[SDV.BBM.BS]Life Sciences [q-bio]/Biochemistry Molecular Biology/Structural Biology [q-bio.BM]ChemistryProtein StabilityOrganic ChemistryHydrogen BondingBridged Bicyclo Compounds HeterocyclicSettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)Protein Structure Tertiary[SDV.BBM.BP]Life Sciences [q-bio]/Biochemistry Molecular Biology/BiophysicsElectrophysiology030104 developmental biologyNicotinic agonistα7 nicotinic receptorEpibatidineLigand-gated ion channel[INFO.INFO-BI]Computer Science [cs]/Bioinformatics [q-bio.QM]medicine.drug
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Modulation of Hippocampal Circuits by Muscarinic and Nicotinic Receptors

2017

This article provides a review of the effects of activation of muscarinic and nicotinic receptors on the physiological properties of circuits in the hippocampal formation. Previous articles have described detailed computational hypotheses about the role of cholinergic neuromodulation in enhancing the dynamics for encoding in cortical structures and the role of reduced cholinergic modulation in allowing consolidation of previously encoded information. This article will focus on addressing the broad scope of different modulatory effects observed within hippocampal circuits, highlighting the heterogeneity of cholinergic modulation in terms of the physiological effects of activation of muscarin…

0301 basic medicinevolume transmissioncholinergic fibersCognitive NeuroscienceNeuroscience (miscellaneous)ReviewHippocampal formationReceptors NicotinicCholinergic modulationHippocampuslcsh:RC321-571tonic depolarization03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineNeuromodulationMuscarinic acetylcholine receptorNeural PathwaysmedicineAnimalsHumanslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCholinergic FibersNicotinic ReceptorsChemistrypresynaptic inhibitionReceptors MuscarinicacetylcholineSensory Systems030104 developmental biologymedicine.anatomical_structureCholinergicNeuroscience030217 neurology & neurosurgeryAcetylcholinemedicine.drugNeuroscienceFrontiers in Neural Circuits
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Effects of cigarette smoking or ingestion of nicotine on platelet 5-hydroxytryptamine (5-HT) levels in smokers and non-smokers.

1992

Platelets of healthy smokers and non-smokers were prepared and their content of 5-hydroxytryptamine was determined by HPLC with electrochemical detection. Platelet 5-HT levels in smokers (728 +/- 156 pmol per 10(8) platelets, mean +/- SEM, n = 9) were significantly higher than those in non-smokers (353 +/- 156 pmol per 10(8) platelets, n = 11). Smoking of a single cigarette caused a transient increase in platelet 5-HT levels by about 350% in non-smokers, but had no additional effect in smokers. Similarly, chewing of nicotine gum (4-8 mg nicotine) resulted in a transient increase in platelet 5-HT by about 100% in non-smokers, but not in smokers. In conclusion, smoking of cigarettes can cause…

AdultBlood Plateletsmedicine.medical_specialtyNicotineSerotoninAdministration OralReceptors NicotinicNicotine03 medical and health sciences0302 clinical medicineCigarette smokingInternal medicineDrug DiscoverymedicineEnterochromaffin CellsIngestionHumansPlateletReceptorGenetics (clinical)5-HT receptorbusiness.industrySmokingGeneral Medicinerespiratory tract diseases3. Good healthEndocrinologyNicotine gum030220 oncology & carcinogenesisReceptors Serotoninbehavior and behavior mechanismsMolecular MedicineSerotoninbusiness030217 neurology & neurosurgerymedicine.drugThe Clinical investigator
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Association of nicotinic acetylcholine receptor subunit alpha 4 polymorphisms with nicotine dependence in 5500 Germans.

2009

Polymorphisms in the CHRNA4 gene coding the nicotinic acetylcholine receptor subunit alpha 4 have recently been suggested to play a role in the determination of smoking-related phenotypes. To examine this hypothesis, we conducted a genetic association study in three large samples from the German general population (N(1)=1412; N(2)=1855; N(3)=2294). Five single-nucleotide polymorphisms in CHRNA4 were genotyped in 5561 participants, including 2707 heavily smoking cases (regularly smoking at least 20 cigarettes per day) and 2399 never-smoking controls (or=100 cigarettes over lifetime). We examined associations of the polymorphisms with smoking case-control status and with the extent of nicotin…

AdultMaleAdolescentGenotypeProtein subunitBiologyPharmacologyReceptors NicotinicPolymorphism Single NucleotideWhite PeopleGermanyGeneticsmedicineHumansGenetic Predisposition to DiseaseNicotine dependenceAgedPharmacologyAged 80 and overTobacco Use DisorderMiddle Agedmedicine.diseaseNicotinic acetylcholine receptorPhenotypeMolecular MedicineFemaleSmoking CessationThe pharmacogenomics journal
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Cellular distribution of nicotinic acetylcholine receptor subunit mRNAs in the human cerebral cortex as revealed by non-isotopic in situ hybridizatio…

1994

The pharmacology of telencephalic nicotinic acetylcholine receptors (nAChRs) has become an important issue in recent years. While in the human brain a direct pharmacological assessment is difficult to achieve the visualization of nAChRs has been enabled by histochemical techniques providing an ever increasing and improving resolution. Receptor autoradiography was used to visualize binding sites on the level of cortical layers whereas immunohistochemistry has allowed for the cell type-specific and ultrastructural localization of receptor protein. Further investigations have to elucidate the cellular sites of NAChR biosynthesis by visualizing subunit-specific transcripts. Using autopsy sample…

AdultMaleIn situ hybridizationBiologyReceptors NicotinicCellular and Molecular NeuroscienceCortex (anatomy)medicineHumansRNA MessengerReceptorMolecular BiologyIn Situ HybridizationAcetylcholine receptorAgedCerebral CortexNeuronsSequence Homology Amino AcidHuman brainRNA ProbesMiddle AgedAlkaline PhosphataseNicotinic acetylcholine receptormedicine.anatomical_structureNicotinic agonistCerebral cortexFemaleNeuroscienceBrain research. Molecular brain research
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Association of attentional network function with exon 5 variations of the CHRNA4 gene

2007

Mutational analyses in xenopus oocyte and mice models indicate that the positive effect of nicotine on attention may be modulated by genetic variations within exon 5 of the alpha4 subunit of the nicotinergic acetylcholine receptor gene CHRNA4. The potential relevance of exon 5 is further emphasized by two recent family-based association studies of nicotine dependence because subgroups of nicotine-dependent subjects are thought to 'self-medicate' attentional deficits with nicotine. We investigated a synonymous single nucleotide polymorphisms (SNP): rs1044396, which has recently been associated with nicotine-dependence, plus two adjacent synonymous SNPs rs1044394 and rs1044393 in exon 5 of n …

AdultMaleNicotineXenopusPosterior parietal cortexSingle-nucleotide polymorphismReceptors NicotinicBiologyModels BiologicalWhite PeopleMiceExonParietal LobeGeneticsmedicineAnimalsHumansAttentionMolecular BiologyOddball paradigmGenetics (clinical)Genetic associationGeneticsSupplementary motor areamedicine.diagnostic_testParietal lobeExonsTobacco Use DisorderGeneral MedicineMagnetic Resonance ImagingRadiographymedicine.anatomical_structureAttention Deficit Disorder with HyperactivityFemaleFunctional magnetic resonance imagingHuman Molecular Genetics
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Risk gene variants for nicotine dependence in the CHRNA5-CHRNA3-CHRNB4 cluster are associated with cognitive performance

2010

Recent studies strongly support an association of the nicotinic acetylcholine receptor gene cluster CHRNA5-CHRNA3-CHRNB4 with nicotine dependence (ND). However, the precise genotype-phenotype relationship is still unknown. Clinical and epidemiological data on smoking behavior raise the possibility that the relevant gene variants may indirectly contribute to the development of ND by affecting cognitive performance in some smokers who consume nicotine for reasons of "cognition enhancement." Here, we tested seven single nucleotide polymorphisms (SNPs) rs684513, rs637137, rs16969968, rs578776, rs1051730, rs3743078, rs3813567 from the CHRNA5-CHRNA3-CHRNB4 gene cluster for association with ND, me…

AdultMaleRiskGenotypeGene ExpressionNerve Tissue ProteinsSingle-nucleotide polymorphismReceptors NicotinicBiologyBioinformaticsPolymorphism Single NucleotideNicotineCellular and Molecular NeuroscienceCognitionGene clustermedicineHumansGenetic Predisposition to DiseaseRNA MessengerRisk factorAlleleGenetic Association StudiesGenetics (clinical)AgedGeneticsChromosomes Human Pair 15Gene Expression ProfilingCHRNA5HaplotypeWechsler ScalesGenetic VariationCognitionTobacco Use DisorderMiddle AgedPsychiatry and Mental healthMultigene Familybiology.proteinFemalemedicine.drugAmerican Journal of Medical Genetics Part B: Neuropsychiatric Genetics
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Blockade of nicotinic and muscarinic receptors facilitates spontaneous migration of human peripheral granulocytes: failure in cystic fibrosis.

2012

Circulating leucocytes express muscarinic (m) and nicotinic (n) receptors and synthesize acetylcholine (ACh) regulating various cell functions. Leucocytes from patients with cystic fibrosis contain less ACh; therefore it was tested whether the regulation of cellular functions like migration differed from healthy volunteers.Peripheral blood (10-20 ml) was used, leucocytes were isolated by Ficoll® gradient and the commercial MIGRATEST® combined with flow cytometric analysis was applied (pore size 3 μm).In the absence of test substances 4900±1800 (n=10) leucocytes migrated within a time period of 2 h. In the presence of tubocurarine (TC, 30 μM) the cell number increased to 7500±2700 [n=10] cor…

AdultMalemedicine.medical_specialtyAdolescentCystic FibrosisBiologyReceptors NicotinicGeneral Biochemistry Genetics and Molecular BiologyCholinergic AntagonistsYoung AdultCell Migration Assays LeukocyteCell MovementInternal medicineMuscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4HumansGeneral Pharmacology Toxicology and PharmaceuticsReceptorChildMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2General MedicineReceptors MuscarinicNicotinic agonistEndocrinologyCholinergicFemaleAcetylcholinemedicine.drugGranulocytesLife sciences
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